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Spexin

A 14-amino-acid galanin-family peptide that is dramatically suppressed in obesity and, when given back to animals, reduces food intake and improves lipid handling.

Also known as NPQ, neuropeptide Q, SPX, C12orf39 product

Animal data onlyRodent or other animal studies. Dose translation to humans is genuinely uncertain.

Solid rodent and fish pharmacology plus strong human correlational data showing spexin falls in obesity and rises after weight loss. No human administration studies at all.

How it works

Spexin was discovered by bioinformatics rather than bioassay — a hidden-Markov-model search for unannotated peptide hormones flagged the C12orf39 gene product. It signals through GALR2 and GALR3 but not GALR1, which matters because GALR1 mediates the orexigenic effects of galanin; spexin therefore behaves as galanin's functional opposite for feeding. In rodents it reduces food intake, delays gastric emptying, decreases long-chain fatty acid uptake by adipocytes and improves glucose tolerance. Human adipose tissue spexin mRNA is the single most downregulated transcript in obesity in some datasets, and circulating levels rise after bariatric surgery. Roles in nociception, reproduction and salt-water balance have also been described, which makes it less clean as a metabolic drug candidate than it first appears.

Targets: GALR2, GALR3, Adipocyte fatty acid uptake

Dosing

ProtocolDoseFrequencyRoute
No human protocol existsNot applicable.not establishedsubcutaneous
  • · Rodent studies use body-weight-scaled daily intraperitoneal dosing, often for two weeks. No human dose has been established and the peptide's short half-life means any naive injectable protocol would be pharmacologically pointless.

Cycling

Not applicable.

Work out your exact syringe units →

Pharmacology

Half-life
Very short — a small unmodified linear peptide with no half-life extension. Not characterised in humans.
Onset
Acute anorectic effects within an hour in rodents; metabolic changes over weeks of repeated dosing.
Routes
subcutaneous, intravenous
Molecule
Endogenous 14-amino-acid amidated neuropeptide (galanin family)
Sequence length
14 amino acids

Handling

Diluent
Bacteriostatic water
Lyophilised
Freezer at -20 C.
Reconstituted
Refrigerated, days only.

Mixing

Research-grade only. The C-terminal amidation matters for activity; non-amidated material is inactive.

Side effects

  • commonUnknown in humansNever administered to people in a published study.
  • commonDelayed gastric emptyingDemonstrated in rodents; would plausibly translate as nausea or fullness in humans.

Do not use if

  • Human use outside research.

Combining it

  • redundantsemaglutideBoth slow gastric emptying and reduce intake; there is no reason to expect additive benefit and good reason to expect additive GI misery.

What to monitor

  • · Serum spexin is a research biomarker only.

Legal status

Research reagent. Not approved anywhere.

References

  • Mirabeau et al. 2007, Genome Research — bioinformatic identification of spexin (preclinical)
  • Walewski et al. 2014, Obesity — spexin is downregulated in human obesity and reduces food intake in rodents (preclinical)

Mechanism in depth

A 14-residue peptide encoded by the C12orf39 gene, acting as a ligand at galanin receptors 2 and 3 but not galanin receptor 1 - a selectivity that matters, because GALR1 signalling drives the appetite-stimulating effects of galanin while GALR2/3 signalling appears to do the opposite. Circulating levels are reported to be lower in obesity.

What usually goes wrong

The human evidence is associative - spexin is lower in people with obesity - and association is being sold as mechanism. Lower spexin in obesity is as consistent with consequence as with cause, and no interventional human trial has tested whether raising it changes anything.

Receptor targets

  • Galanin receptor 2 and 3Selective over GALR1

    Reduced food intake and altered lipid handling in animal models

Genuinely uncertain

  • No human interventional data exists.
  • Whether low spexin in obesity is causal or consequent is untested.
  • No validated human dose exists by any route.